Hostname: page-component-cb9f654ff-hn9fh Total loading time: 0 Render date: 2025-09-02T23:08:23.566Z Has data issue: false hasContentIssue false
  • English
  • Français

Distribution of ABO and Rhesus Blood Groups in G6PD Deficient Chinese and Malay Newborns

Published online by Cambridge University Press:  01 August 2014

N. Saha ,
B.R. Hawkins  and
H.B. Wong
N. Saha*
Affiliation:
Department of Physiology, University of Singapore, Singapore Department of Human Biology, The John Curtin School of Medical Research, Australian National University, Canberra, Australia
B.R. Hawkins
Affiliation:
Department of Human Biology, The John Curtin School of Medical Research, Australian National University, Canberra, Australia
H.B. Wong
Affiliation:
Department of Paediatrics, University of Singapore, Singapore
*
Department of Physiology, Faculty of Medicine, University of Khartoum, P.O. Box 102, Khartoum, Sudan

Abstract

Core share and HTML view are not available for this content. However, as you have access to this content, a full PDF is available via the ‘Save PDF’ action button.

The distribution of ABO blood groups was studied in 459 Chinese and 65 Malay newborns with deficiency of G6PD, and in 1181 Chinese and 535 Malay newborns with normal levels of the enzyme. Similarly, the distribution of Rhesus blood groups was studied in 248 G6PD deficient Chinese newborns and in 255 normal subjects. No association between the ABO blood groups and G6PD deficiency was observed in either Chinese or Malays. For the Rhesus system there was found to be a statistically significant decrease in the frequency of genotypes containing the complex R 1 in G6PD deficient subjects compared with that in normal subjects.

Information

Type
Research Article
Information
Acta geneticae medicae et gemellologiae: twin research , Volume 24 , Issue 1-2 , January 1975 , pp. 131 - 135
Copyright
Copyright © The International Society for Twin Studies 1975

References

REFERENCES

Adam, A., Dressler, L., Sheba, C., Szeinberg, A. 1963. A comparative study of blood group distribution among normal and G-6-PD deficient subjects in Israel. 11th Int. Congr. of Genetics, The Hague.Google Scholar
Motulsky, A.G., Campbell-Kraut, J.M. 1961. Population genetics of glucose-6-phosphate dehydrogenase deficiency of the red cell. In Blumberg, B.S. (ed.): Proceedings of the Conference on Genetic Polymorphisms and Geographic Variations in Disease (p. 159). New York: Grune and Stratton.Google Scholar
Saha, N., Banerjee, B. 1971 a. Erythrocyte G-6-PD deficiency among Chinese and Malays of Singapore. Trop. Geogr. Med., 23: 141–144.Google Scholar
Saha, N., Benerjee, B. 1971 b. Incidence of glucose-6-phosphate dehydrogenase deficiency among different ethnic groups of India. Hum. Hered., 21: 78–82.Google Scholar
Saha, N., Wong, H.B. 1971. Distribution of ABO and Rh blood groups among G-6-PD deficient and normal Chinese newborns. 4th Int. Congr. of Human Genetics, Paris.Google Scholar
Tarlov, A.R., Brewer, G.J., Carson, P.R., Alving, A.S. 1962. Primaquine sensitivity. Arch. Intern, Med., 109: 209–235.Google Scholar
WHO 1967. Standardisation of procedure for the study of glucose-6-phosphate dehydrogenase. Technical Report Series No. 366.Google Scholar
Woolf, B. 1955. On estimating the relation between blood group and disease. Ann. Hum. Genet., 19: 251–253.Google Scholar